Search results for "genetics [Transcriptome]"

showing 10 items of 3033 documents

Direct conversion of human fibroblast to hepatocytes using a single inducible polycistronic vector

2019

Abstract Background Human fibroblasts can be reprogrammed into induced hepatocyte-like cells through the expression of a set of transcription factors. Although the generation of induced hepatocyte-like cells by HNF4A, HNF1A, and FOXA3 expression has proven to be a robust experimental strategy, using multiple lentivirus results in a highly variable heterogeneous population. Methods We designed and implemented a novel approach based on the delivery of reprogramming factors and green fluorescent protein in a single doxycycline-inducible lentiviral vector using 2A self-cleaving peptides. Results Fibroblasts infected with the lentiviral vector can be amplified in basic fibroblast culture media i…

Male0301 basic medicineInducibleGenetic VectorsGreen Fluorescent ProteinsMedicine (miscellaneous)Biochemistry Genetics and Molecular Biology (miscellaneous)Cell LineViral vectorGreen fluorescent proteinlcsh:BiochemistryMice03 medical and health sciences0302 clinical medicinePolycistronic vectorsmedicineAnimalsHumanslcsh:QD415-436TransgenesFibroblastGeneTranscription factorlcsh:R5-920ChemistryResearchReprogrammingDermisCell BiologyFibroblastsCellular ReprogrammingCell biologyInduced hepatocyte-like cellsiHEPPhenotype030104 developmental biologymedicine.anatomical_structureGenes030220 oncology & carcinogenesisDoxycyclineHepatocytesMolecular MedicineFOXA3Stem celllcsh:Medicine (General)ReprogrammingStem Cell Research & Therapy
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Drosophila Acquires a Long-Lasting Body-Size Memory from Visual Feedback

2019

Summary Grasping an object or crossing a trench requires the integration of information on the operating distance of our limbs with precise distance estimation. The reach of our hands and step size of our legs are learned by the visual feedback we get during our actions. This implicit knowledge of our peripersonal space is first acquired during infancy but will be continuously updated throughout our whole life [ 1 ]. In contrast, body size of holometabolous insects does not change after metamorphosis; nevertheless, they do have to learn their body reaches at least once. The body size of Drosophila imagines can vary by about 15% depending on environmental factors like food quality and temper…

Male0301 basic medicineMemory Long-Termmedia_common.quotation_subjectBiologyGeneral Biochemistry Genetics and Molecular BiologyMemorizationMotion (physics)03 medical and health sciences0302 clinical medicineFeedback SensoryAnimalsBody SizeContrast (vision)Computer visionSet (psychology)media_commonLong-term memorybusiness.industryDrosophila melanogaster030104 developmental biologyClimbingVisual PerceptionMemory consolidationArtificial intelligenceGeneral Agricultural and Biological SciencesbusinessParallaxPhotic Stimulation030217 neurology & neurosurgeryCurrent Biology
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Cross-sectional associations between angiotensin-converting enzyme inhibitor use and cancer diagnosis in US adults

2020

The objective of the present study was to investigate the association between angiotensin-converting enzyme (ACE) inhibitor use and cancer incidence (overall, and breast, prostate, and colorectal cancers specifically) in a large representative sample of US adults. Cross-sectional data on cancer diagnosis, timing of cancer diagnosis, ACE inhibitor use, and other characteristics were extracted from 49 512 adults aged ≥ 20 years participating in the National Health and Nutrition Examination Survey (1999–2016). Multivariable-logistic and propensity score matching (PSM) regressions examined the relationship between pre-diagnosis use of ACE inhibitors and diagnosis of all cancers, and breast, pro…

Male0301 basic medicineOncologyEpidemiology[SDV]Life Sciences [q-bio]Angiotensin-Converting Enzyme InhibitorsAngiotensin-converting enzyme inhibitors Cancer · Epidemiology NHANES · ObservationalProstate cancer0302 clinical medicineRisk FactorsSurveys and QuestionnairesObservationalCancerAged 80 and over2. Zero hungerbiologyIncidenceGeneral MedicineMiddle Aged3. Good health030220 oncology & carcinogenesisFemaleColorectal Neoplasmsmedicine.drugAdultmedicine.medical_specialtyNational Health and Nutrition Examination SurveyBreast NeoplasmsGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineNHANESHumansAgedbusiness.industryProstatic NeoplasmsCancerAngiotensin-converting enzymeOdds ratioAngiotensin-converting enzyme inhibitors Cancer Epidemiology NHANES Observationalmedicine.diseaseUnited StatesCross-Sectional StudiesLogistic Models030104 developmental biologyPropensity score matchingACE inhibitorbiology.proteinbusinessBody mass indexClinical and Experimental Medicine
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Genome-Wide Association Study for Incident Myocardial Infarction and Coronary Heart Disease in Prospective Cohort Studies: The CHARGE Consortium

2016

Background Data are limited on genome-wide association studies (GWAS) for incident coronary heart disease (CHD). Moreover, it is not known whether genetic variants identified to date also associate with risk of CHD in a prospective setting. Methods We performed a two-stage GWAS analysis of incident myocardial infarction (MI) and CHD in a total of 64,297 individuals (including 3898 MI cases, 5465 CHD cases). SNPs that passed an arbitrary threshold of 5×10−6 in Stage I were taken to Stage II for further discovery. Furthermore, in an analysis of prognosis, we studied whether known SNPs from former GWAS were associated with total mortality in individuals who experienced MI during follow-up. Res…

Male0301 basic medicineOncologyPREDICTIONMyocardial Infarctionlcsh:MedicineGenome-wide association studyCoronary Artery DiseaseCardiovascular MedicineSUSCEPTIBILITY030204 cardiovascular system & hematologyCardiovascularBioinformaticsincident myocardial infarctionCohort StudiesCoronary artery diseaseMathematical and Statistical Techniques0302 clinical medicineDESIGNMedicine and Health Sciences2.1 Biological and endogenous factorsProspective StudiesMyocardial infarctionAetiologyCooperative Behaviorlcsh:ScienceProspective cohort studyRISKscreening and diagnosisMultidisciplinaryResearch Support Non-U.S. Gov'tSingle NucleotideGenomicsMiddle Aged3. Good healthMultidisciplinary SciencesDetectionCHROMOSOME 9P21Heart DiseaseResearch DesignCardiovascular DiseasesCARDIOVASCULAR-DISEASEPhysical SciencesScience & Technology - Other TopicsFemaleStatistics (Mathematics)4.2 Evaluation of markers and technologiesResearch ArticleCohort studymedicine.medical_specialtyGeneral Science & TechnologyCardiologySingle-nucleotide polymorphismResearch and Analysis MethodsGenome ComplexityPolymorphism Single Nucleotide03 medical and health sciencescoronary hearth diseaseInternal medicineMD MultidisciplinaryGenome-Wide Association StudiesGeneticsJournal ArticlemedicineHumansSNPGenetic Predisposition to DiseasePolymorphismStatistical MethodsHeart Disease - Coronary Heart DiseaseMETAANALYSISAgedGenetic associationta112Science & Technologybusiness.industryPreventionlcsh:RHuman GenomeBiology and Life SciencesComputational BiologyHuman GeneticsGenome AnalysisROTTERDAMmedicine.diseaseIntronsGood Health and Well Being030104 developmental biologyGenetic LociGenetics of Diseaselcsh:Q3111 BiomedicinebusinessMathematicsMeta-AnalysisGenome-Wide Association StudyPLOS ONE
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Mesenchymal stem cells derived from inflamed dental pulpal and gingival tissue: a potential application for bone formation

2017

Background Chronic periodontal disease is an infectious disease consisting of prolonged inflammation of the supporting tooth tissue and resulting in bone loss. Guided bone regeneration procedures have become common and safe treatments in dentistry, and in this context dental stem cells would represent the ideal solution as autologous cells. In this study, we verified the ability of dental pulp mesenchymal stem cells (DPSCs) and gingival mesenchymal stem cells (GMSCs) harvested from periodontally affected teeth to produce new mineralized bone tissue in vitro, and compared this to cells from healthy teeth. Methods To characterize DPSCs and GMSCs, we assessed colony-forming assay, immunophenot…

Male0301 basic medicinePathologyCellular differentiationGingivaMedicine (miscellaneous)Bone tissue0302 clinical medicineOsteogenesisMedicinelcsh:QD415-436Pulpal and gingival mesenchymal stem cellsCells CulturedStem cell transplantation for articular cartilage repairlcsh:R5-920Heat shock proteinCell DifferentiationADFsMiddle AgedGingivitismedicine.anatomical_structureBone formationMolecular MedicineFemaleStem celllcsh:Medicine (General)Adultmedicine.medical_specialtyAdolescentBiochemistry Genetics and Molecular Biology (miscellaneous)Proinflammatory cytokinelcsh:Biochemistry03 medical and health sciencesstomatognathic systemHumansPeriodontitisBone regenerationDental PulpAgedProinflammatory cytokinesInflammationbusiness.industryResearchMesenchymal stem cellMesenchymal Stem Cells030206 dentistryCell BiologyDental diseaseInflammation Dental disease Pulpal and gingival mesenchymal stem cells Bone formation Heat shock protein ADFs Proinflammatory cytokinesstomatognathic diseases030104 developmental biologyCancer researchPulp (tooth)businessStem Cell Research & Therapy
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Comprehensive platelet phenotyping supports the role of platelets in the pathogenesis of acute venous thromboembolism - results from clinical observa…

2020

Background: The pathogenesis of arterial and venous thrombosis is in large part interlaced. How much platelet phenotype relates to acute venous thromboembolism (VTE) independent of the underlying cardiovascular profile is presently poorly investigated.Methods: Platelet count and mean platelet volume (MPV), platelet aggregation in whole blood and platelet rich plasma (PRP), platelet-dependent thrombin generation (TG) and platelet surface activation markers were measured under standardized conditions. Machine learning was applied to identify the most relevant characteristics associated with VTE from a large array (N = 58) of clinical and plateletrelated variables.Findings: VTE cases (N = 159)…

Male0301 basic medicinePlatelet Aggregationlcsh:MedicineDETERMINANTSGastroenterologyMachine LearningPathogenesisACTIVATION0302 clinical medicineRisk FactorsPlateletWhole bloodlcsh:R5-920AspirinOUTCOMESThrombinVenous ThromboembolismGeneral MedicineMiddle AgedThrombosisVenous thrombosis030220 oncology & carcinogenesisAcute DiseaseFemaleDisease Susceptibilitylcsh:Medicine (General)Research Papermedicine.drugBlood Plateletsmedicine.medical_specialtyPlatelet Function TestsGeneral Biochemistry Genetics and Molecular BiologyImmunophenotyping03 medical and health sciencesACUTE PULMONARY-EMBOLISMRISK-FACTORInternal medicinemedicineHumansMean platelet volumeMETAANALYSISAgedPlatelet Countbusiness.industrylcsh:RPlatelet Activationmedicine.diseasePREVENTIONASPIRINTHROMBOSIS030104 developmental biologyPlatelet-rich plasmaVOLUMEbusinessBiomarkersEBioMedicine
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Inhibition of histone deacetylation rescues phenotype in a mouse model of Birk-Barel intellectual disability syndrome

2020

Mutations in the actively expressed, maternal allele of the imprinted KCNK9 gene cause Birk-Barel intellectual disability syndrome (BBIDS). Using a BBIDS mouse model, we identify here a partial rescue of the BBIDS-like behavioral and neuronal phenotypes mediated via residual expression from the paternal Kcnk9 (Kcnk9pat) allele. We further demonstrate that the second-generation HDAC inhibitor CI-994 induces enhanced expression from the paternally silenced Kcnk9 allele and leads to a full rescue of the behavioral phenotype suggesting CI-994 as a promising molecule for BBIDS therapy. Thus, these findings suggest a potential approach to improve cognitive dysfunction in a mouse model of an impri…

Male0301 basic medicinePotassium Channels[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyGeneral Physics and AstronomyDiseasePhenylenediamines[SDV.BBM.BM] Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCraniofacial AbnormalitiesHistonesMice0302 clinical medicineIntellectual disabilityImprinting (psychology)lcsh:ScienceMice KnockoutGeneticsMultidisciplinaryBehavior AnimalbiologyNeurodevelopmental disordersDevelopmental disordersQBrainPhenotypeUp-RegulationPhenotypeHistoneGene Knockdown TechniquesBenzamidesMuscle HypotoniaFemaleLocus CoeruleusEpigeneticsScienceArticleGeneral Biochemistry Genetics and Molecular BiologyGenomic Imprinting03 medical and health sciencesDevelopmental disorders ; Neurodevelopmental disorders ; EpigeneticsIntellectual DisabilitymedicineAnimalsHumansddc:610AlleleGene[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyGeneral Chemistrymedicine.diseaseHistone Deacetylase InhibitorsMice Inbred C57BLDisease Models Animal030104 developmental biologyAcetylationMutationbiology.proteinlcsh:Q030217 neurology & neurosurgery
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Lipidomic profiling identifies signatures of metabolic risk

2020

Background: Metabolic syndrome (MetS), the clustering of metabolic risk factors, is associated with cardiovascular disease risk. We sought to determine if dysregulation of the lipidome may contribute to metabolic risk factors. Methods: We measured 154 circulating lipid species in 658 participants from the Framingham Heart Study (FHS) using liquid chromatography-tandem mass spectrometry and tested for associations with obesity, dysglycemia, and dyslipidemia. Independent external validation was sought in three independent cohorts. Follow-up data from the FHS were used to test for lipid metabolites associated with longitudinal changes in metabolic risk factors. Results: Thirty-nine lipids were…

Male0301 basic medicineResearch paperdhSL dihydrosphingolipidBMI body mass indexlcsh:MedicineATHEROGENIC LIPOPROTEINSBioinformaticsFHS Framingham Heart StudyPC phosphatidylcholinePESA Progression of Early Subclinical Atherosclerosis0302 clinical medicineFramingham Heart StudyRisk FactorsSAFHS San Antonio Family Heart StudyLC-MS/MS liquid chromatography-tandem mass spectrometryMedicineLongitudinal StudiesMetabolic riskPLASMA SPHINGOLIPID METABOLISMPOPULATIONlcsh:R5-920education.field_of_studySPHINGOMYELINCE cholesteryl esterDysglycemiaGeneral MedicineMiddle AgedLipidomePS phosphatidylserineCardiovascular diseaseLipidsMetabolic syndrome3. Good healthCARDIOVASCULAR-DISEASE030220 oncology & carcinogenesisHEARTMetS metabolic syndromeFemaleDisease SusceptibilityLGPL lysoglycerophospholipidSL sphingolipidlcsh:Medicine (General)LPE lysophosphatidylethanolamineAdultFDR false discovery rateTAG triacylglycerolPopulationCer ceramideCVD cardiovascular diseasePE phosphatidylethanolamineDENSITY-LIPOPROTEINRisk AssessmentGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesAnimalsHumanseducationT2DM type II diabetes mellitusAgedLPC lysophosphatidylcholineSM sphingomyelinbusiness.industryCERAMIDElcsh:RHDL-C High density lipoprotein cholesterolBiomarkerLipid Metabolismmedicine.diseaseObesitySphingolipidCross-Sectional Studies030104 developmental biologyDyslipidemiaERF Erasmus Family StudyLipidomicsMetabolic syndromeINDUCED INSULIN-RESISTANCEbusinessDAG diacylglycerolBody mass indexBiomarkersDyslipidemiaMRM multiple reaction monitoringFASTING GLUCOSE
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Evolution of the immune landscape during progression of pancreatic intraductal papillary mucinous neoplasms to invasive cancer

2020

ABSTRACT: Background: Intraductal papillary mucinous neoplasms (IPMNs) are precursor lesions of pancreatic cancer, which is characterized by an immunosuppressive microenvironment. Yet, the spatial distribution of the immune infiltrate and how it changes during IPMN progression is just beginning to be understood. Methods: We obtained tissue samples from patients who underwent pancreatic surgery for IPMN, and performed comprehensive immunohistochemical analyses to investigate the clinical significance, composition and spatial organization of the immune microenvironment during progression of IPMNs. Survival analysis of pancreatic cancer patients was stratified by tumour infiltrating immune cel…

Male0301 basic medicineResearch paperendocrine system diseasesT cellPancreatic Intraductal Neoplasmslcsh:MedicineGeneral Biochemistry Genetics and Molecular BiologyMalignant transformation03 medical and health sciences0302 clinical medicineImmune systemStromaT-Lymphocyte SubsetsPancreatic cancerTumor MicroenvironmentPremalignant lesionHumansMedicineNeoplasm InvasivenessAgedTumour microenvironmentlcsh:R5-920Intraductal papillary mucinous neoplasmIntraductal papillary mucinous neoplasmbusiness.industrylcsh:RCancerPancreatic cancerGeneral MedicineMiddle Agedmedicine.disease030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchTumour immunologyFemalelcsh:Medicine (General)businessCD8EBioMedicine
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Serotonin modulates a depression-like state in Drosophila responsive to lithium treatment

2016

Major depressive disorder (MDD) affects millions of patients; however, the pathophysiology is poorly understood. Rodent models have been developed using chronic mild stress or unavoidable punishment (learned helplessness) to induce features of depression, like general inactivity and anhedonia. Here we report a three-day vibration-stress protocol for Drosophila that reduces voluntary behavioural activity. As in many MDD patients, lithium-chloride treatment can suppress this depression-like state in flies. The behavioural changes correlate with reduced serotonin (5-HT) release at the mushroom body (MB) and can be relieved by feeding the antidepressant 5-hydroxy-L-tryptophan or sucrose, which …

Male0301 basic medicineSerotoninSucroseLithium (medication)ScienceGeneral Physics and AstronomyLearned helplessnessWalkingLithiumMotor ActivityVibrationArticleGeneral Biochemistry Genetics and Molecular Biology5-Hydroxytryptophan03 medical and health sciences0302 clinical medicineStress PhysiologicalCyclic AMPAnimalsMedicineReceptorMultidisciplinaryDepressionbusiness.industryQfungiBrainAnhedoniaGeneral Chemistrymedicine.diseaseImmunohistochemistryAntidepressive Agents3. Good healthDrosophila melanogaster030104 developmental biologyReceptors SerotoninMushroom bodiesAntidepressantMajor depressive disorderFemaleSerotoninmedicine.symptombusinessNeuroscience030217 neurology & neurosurgerySignal Transductionmedicine.drugNature Communications
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